MEMORY LOSS: THERE IS NO CURE. THERE IS PREVENTION.

Treatment of Alzheimer's disease (AD) is complicated by the lack of knowledge of its pathogenesis.

Scientists have concluded that curcumin extract (not to be confused with turmeric) improves brain function and reduces the risk of developing brain diseases, being effective in treating Alzheimer's.

Many people with age suffer from Alzheimer's disease. This is a chronic neurodegenerative disease characterized by progressive memory loss. Therefore, it is important to be able to prevent this disease in youth. First, for this, doctors advise to train memory, learn foreign languages, developing brain abilities. However, it is also important to replenish the body with substances whose reserves are depleted with age.

First of all, curcumin increases the level of a hormone, the decrease of which leads to common brain disorders (including depression and Alzheimer's disease). That is, curcumin helps the body maintain its natural functions in good condition.

Memory loss.

An animal study showed that a noticeable effect was found in animals that received curcumin for 12 weeks. In addition, the study confirmed that curcumin can significantly improve cognitive function in elderly animals.

Unfortunately, there is no effective treatment for Alzheimer's disease yet. Therefore, preventing its occurrence in the first place is extremely important. It is known that inflammation and oxidative damage play a role in Alzheimer's disease, and curcumin neutralizes them. Moreover, a key feature of Alzheimer's disease is the accumulation of amyloid plaques. Studies show that curcumin will help clear these plaques.

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AD patients have defects in the phagocytosis of beta-amyloid (1-42) (Abeta) in vitro by innate immune cells, monocytes/macrophages, and Abeta plaque clearance.

The natural product curcuminoids increased Abeta clearance from the brain in animal models. Thus, we treated macrophages from six asthma patients. As well as three control patients with curcuminoids in vitro. Abeta uptake was measured by fluorescence and confocal microscopy.

The baseline Abeta uptake intensity by AD macrophages was significantly lower compared to control macrophages, involving surface binding but not intracellular uptake. However, after treatment of macrophages with curcuminoids, Abeta uptake by macrophages from three of the six asthma patients was significantly increased (P<0.001 to 0.081). That is, confocal microscopy Moreover, curcuminoid treatment showed co-localization with phalloidin in the intracellular compartment even after treatment.

Finally, immunomodulation of innate immunity with curcuminoids is a safe approach to immune clearance of amyloidosis in the brain with bronchial asthma.